My theory is big gobs of syncytia-joined-cells plus extracellular matrix jumble via matrix metalloproteases snipping things off plus netosis byproducts / fibrin. Triggered by the multi-arginine motif that echoes decades of work with HIV-1 Tat protein as a gene transfection tool - makes its cargo head straight for the nucleus and break past protective borders. Tat causes Syncytia. This motif magically appeared in Spike and has been discussed as the furin cleavage site. Spike causes Syncytia within several hours.
At least in parts of it. In the lab the syncytia start building within 4 hours, and then death of cells involved in the syncytia starts gaining at 48 hours. I wonder, what would a mass of fibrin + immune defense + building cell blobs look like histologically?
So that's interesting. Seems not too weird that platelets would be a bit perturbed by spike and it's too bad they don't use any other kind of molecule to compare results. Also they speculate that it's because of RGD403 on the spike, but who can say for sure - however, that D is one of the residues of interest that changed with BA.2/4/5. Maybe that makes for less problems.
So it's a good thing the bivalent booster kept the RGD-having version for very clear, not-suspicious reasons.
Agreed, it's certainly something to look into more. Coagulation isn't my main focus at the moment, but I hope it helped. I'll keep reading, you got good stuff :)
Marc Girardot of Covid Myth Buster substack (and his bolus theory ) theorises that they’re elastin, from the damaged endothelium - but I’m not sure that sounds biologically feasible?
Simultaneously, if he is so wedded to the idea that "bolus" applies to all vaccines, why would the clots be only seen now? Humans, and elderly, have been vaxxed up like crazy for the last two decades. So I think the only approach he can really take with the clots is to call them fake, not to try to explain them with his theory.
Nah. I was in PANDA with him for a year. I don't give incredibly disingenuous people respect. I've interacted with him way more than most people though, and I'm pretty biased against him. so take my opinion for what it's worth, close to nothing without evidence (that I'm way too lazy to accumulate lol)
I wasn't saying they don't exist but that I expected a lot more people to give a hint of something strange happening, even if they didn't want to directly talk about the clots. People I contacted talked about Plandemics and death certs issued incorrectly so these types of people would have been open to talking about clots but none of them had seen anything. And this was over a quite wide geographic area.
I want to hear from actual people who have relatives who've died and they've pulled these things out. Ideally they sent them off to the lab for testing but unlikely. So far, it's just YouTube videos of a handful of doctors on the other side of the pond. So I'm still unsure what to believe.
The live blood stuff usually focuses on the rouleaux of red blood cells - but Cole's samples are apparently acellular. So if his samples are just synthetic fakes then the rouleaux could definitely still be valid and relevant to sudden deaths. As far as I can see there's no way to assign a chain of authenticity to any of this at the moment, just have to keep everything tentative.
Shoo Brian! You're supposed to be writing about how the variants are not variants of virus but variants of labs! I think...
At first the shock of seeing those calamari-like structures was very concerning, but after watching Died Suddenly I actually have a few questions of the structures. One comment I've raised is to how exactly these extensive, giant structures were formed without causing noticeable changes in pathology. Would we see pervasive blood pressure elevation? In which case that wouldn't inherently be a "Died Suddenly" incident if that would indicate one would be predisposed to cardiovascular issues.
I intended to write a post on Parkinson's but I became a bit exhausted on writing Anthology Series posts. I may revisit it given the circumstances, but parkinsonism and viral parkinsonism in particular is actually known and documented in the literature. To what extent the vaccines could be contributing to it is unknown, however there's evidence that this isn't an inherently new phenomenon. It's another one of those concerning things that may not be too out of the ordinary, to say the least.
If they are in arteries, they are akin to a one-way tourniquet, I suppose, theoretically. But I really have my doubts that these things are real at the moment. Wouldn't there be like a flood of stuff from outside the anglosphere, where the mRNA has also been used?
Just curious, how much of the world outside the anglosphere prefers cremation over embalming? I keep hearing stories about "crematoriums" being overrun in China, for example.
I think what I find strange is that the structures should probably lead to some harm, or sudden death before they become these giant, foot long worms. We know microclots and damage to microvasculature in extremities is occurring, but are these people not experiencing this and then suddenly dropping dead from the calamari strings?
I think the ambiguity and the lack of information is what drives some of the appeal to these things. It has that shock horror, it adds to this notion that the vaccines are intended to kill everyone, and so it just works to substantiate people's biases. But again, the more I think about it the more skeptical I become of these things in particular, especially given that we know clots are occurring.
Right, but the Pretorius spike+fibrin>amyloid-fibrinolysis-resistant-fibrin angle has seemed like a robust template for building theories why the vaxxed have resilient "structures" of whatever type. If it's simply fibrin, why isn't it breaking down as all fibrin/clots do?
Walter M Chesnut wrote that part of the spike coded for fibrin - it was a while ago and I cant remember in which post he said it and not being an actual scientist if its true
I can't claim even a partial understanding of clotting. But fibrin is a product of multiple glycoproteins generated, processed, and interlinked, so I don't think there can be much meaningful similarity.
What I do know (not really, but have written down from Pretorius videos) is that when clots form they are supposed to incorporate a breakdown factor, which is plasminogen, and presumably spike protein is disrupting this protein or its ability to work. It's a bit annoying that more labs aren't pursuing this angle. So anyway that part could possibly still be happening even without the amyloid bit. Maybe the amyloid effect is an artifact of working with spike in the bloodflow simulator but in the body you just get breakdown/plasminogin-defective but non-amyloid clots.
downregulation of "x" seems to crop up frequently in the research papers I've skimmed.
Like many, I was traumatised by the videos but reflecting on Ryan Coles lack of urgency on analysing the clots and lack of real world observations I'm starting to lean towards the naked emporers (Ivor Cumins) viewpoint that there's either fakery or it's not as frequent as some people would have us believe. Either way it needs investigating.
Perhaps. Martenson also does a back of envelope fibrin - to - blood volume calculation but that's in the assumption of post-mortem pooling, not of circulation + buildup.
My theory is big gobs of syncytia-joined-cells plus extracellular matrix jumble via matrix metalloproteases snipping things off plus netosis byproducts / fibrin. Triggered by the multi-arginine motif that echoes decades of work with HIV-1 Tat protein as a gene transfection tool - makes its cargo head straight for the nucleus and break past protective borders. Tat causes Syncytia. This motif magically appeared in Spike and has been discussed as the furin cleavage site. Spike causes Syncytia within several hours.
Perhaps. One might expect the histology to look more akin to a placenta, a sort of mashup of nuclei and cellular parts, though
At least in parts of it. In the lab the syncytia start building within 4 hours, and then death of cells involved in the syncytia starts gaining at 48 hours. I wonder, what would a mass of fibrin + immune defense + building cell blobs look like histologically?
Dr. McCullough just wrote about a recently published article that picks up on some of the stuff I've been looking into the last months including syncytia and cell penetrating peptides - https://petermcculloughmd.substack.com/p/pathological-syncytia-formation-with
Spike aside, nattokinase = generally good stuff. Worth developing a taste for natto.
Unfortunately for me I am allergic to Soy or I would be taking the supplements.
I keep forgetting to drop in at Mitsuwa to roll-play Japanese Working Class Lunch with some in the dining section! It's on my to-do list.
So they are normal sometimes at least (if not all the time)?
Next lead:
https://www.biorxiv.org/content/10.1101/2022.11.22.517574v1
So that's interesting. Seems not too weird that platelets would be a bit perturbed by spike and it's too bad they don't use any other kind of molecule to compare results. Also they speculate that it's because of RGD403 on the spike, but who can say for sure - however, that D is one of the residues of interest that changed with BA.2/4/5. Maybe that makes for less problems.
So it's a good thing the bivalent booster kept the RGD-having version for very clear, not-suspicious reasons.
Agreed, it's certainly something to look into more. Coagulation isn't my main focus at the moment, but I hope it helped. I'll keep reading, you got good stuff :)
Missed that one - thank you, will take a look
Marc Girardot of Covid Myth Buster substack (and his bolus theory ) theorises that they’re elastin, from the damaged endothelium - but I’m not sure that sounds biologically feasible?
Simultaneously, if he is so wedded to the idea that "bolus" applies to all vaccines, why would the clots be only seen now? Humans, and elderly, have been vaxxed up like crazy for the last two decades. So I think the only approach he can really take with the clots is to call them fake, not to try to explain them with his theory.
good point, why now
Omg Giradot is everywhere. He will never acknowledge a single point you make, I assure you. There is no reasoning with him
Hey, he's been on some boats, he's seen fog dissipate, you got to give the guy respect where it's due.
Nah. I was in PANDA with him for a year. I don't give incredibly disingenuous people respect. I've interacted with him way more than most people though, and I'm pretty biased against him. so take my opinion for what it's worth, close to nothing without evidence (that I'm way too lazy to accumulate lol)
I wasn't saying they don't exist but that I expected a lot more people to give a hint of something strange happening, even if they didn't want to directly talk about the clots. People I contacted talked about Plandemics and death certs issued incorrectly so these types of people would have been open to talking about clots but none of them had seen anything. And this was over a quite wide geographic area.
I want to hear from actual people who have relatives who've died and they've pulled these things out. Ideally they sent them off to the lab for testing but unlikely. So far, it's just YouTube videos of a handful of doctors on the other side of the pond. So I'm still unsure what to believe.
Unsure here as well.
I’m no scientist but I would think that having these clots analyzed by a decent laboratory would give us the answers.....
Apparently the human species in the 21st Century is categorically incapable of inspecting its own dead. Just can't do it! because reasons. ¯\_(ツ)_/¯
something not right.
What about the live blood analysis people claiming the clots are some kind of hydrogel? https://anamihalceamdphd.substack.com/p/breaking-news-c19-vials-ribbons-are?utm_source=substack&utm_medium=email I haven't seen anyone try to refute that, it seems to just be ignored in the more mainstream alt-covid substances.
The live blood stuff usually focuses on the rouleaux of red blood cells - but Cole's samples are apparently acellular. So if his samples are just synthetic fakes then the rouleaux could definitely still be valid and relevant to sudden deaths. As far as I can see there's no way to assign a chain of authenticity to any of this at the moment, just have to keep everything tentative.
Shoo Brian! You're supposed to be writing about how the variants are not variants of virus but variants of labs! I think...
At first the shock of seeing those calamari-like structures was very concerning, but after watching Died Suddenly I actually have a few questions of the structures. One comment I've raised is to how exactly these extensive, giant structures were formed without causing noticeable changes in pathology. Would we see pervasive blood pressure elevation? In which case that wouldn't inherently be a "Died Suddenly" incident if that would indicate one would be predisposed to cardiovascular issues.
I intended to write a post on Parkinson's but I became a bit exhausted on writing Anthology Series posts. I may revisit it given the circumstances, but parkinsonism and viral parkinsonism in particular is actually known and documented in the literature. To what extent the vaccines could be contributing to it is unknown, however there's evidence that this isn't an inherently new phenomenon. It's another one of those concerning things that may not be too out of the ordinary, to say the least.
If they are in arteries, they are akin to a one-way tourniquet, I suppose, theoretically. But I really have my doubts that these things are real at the moment. Wouldn't there be like a flood of stuff from outside the anglosphere, where the mRNA has also been used?
Just curious, how much of the world outside the anglosphere prefers cremation over embalming? I keep hearing stories about "crematoriums" being overrun in China, for example.
No idea. It's a funny feature of modern life, that humans have very little clue how the whole death thing is really handled.
I think what I find strange is that the structures should probably lead to some harm, or sudden death before they become these giant, foot long worms. We know microclots and damage to microvasculature in extremities is occurring, but are these people not experiencing this and then suddenly dropping dead from the calamari strings?
I think the ambiguity and the lack of information is what drives some of the appeal to these things. It has that shock horror, it adds to this notion that the vaccines are intended to kill everyone, and so it just works to substantiate people's biases. But again, the more I think about it the more skeptical I become of these things in particular, especially given that we know clots are occurring.
my input on this topic can be found here:
https://mejbcart.substack.com/p/stew-peters-documentary-died-suddenly
https://mejbcart.substack.com/p/where-did-the-iron-go-the-dopamine
for anyone interested..
Not amyloid. Fibrin
Would that be as a result of the gene therapies? I can't in good conscience call them "vaccines" when they technically are not.
Right, but the Pretorius spike+fibrin>amyloid-fibrinolysis-resistant-fibrin angle has seemed like a robust template for building theories why the vaxxed have resilient "structures" of whatever type. If it's simply fibrin, why isn't it breaking down as all fibrin/clots do?
Walter M Chesnut wrote that part of the spike coded for fibrin - it was a while ago and I cant remember in which post he said it and not being an actual scientist if its true
I can't claim even a partial understanding of clotting. But fibrin is a product of multiple glycoproteins generated, processed, and interlinked, so I don't think there can be much meaningful similarity.
What I do know (not really, but have written down from Pretorius videos) is that when clots form they are supposed to incorporate a breakdown factor, which is plasminogen, and presumably spike protein is disrupting this protein or its ability to work. It's a bit annoying that more labs aren't pursuing this angle. So anyway that part could possibly still be happening even without the amyloid bit. Maybe the amyloid effect is an artifact of working with spike in the bloodflow simulator but in the body you just get breakdown/plasminogin-defective but non-amyloid clots.
downregulation of "x" seems to crop up frequently in the research papers I've skimmed.
Like many, I was traumatised by the videos but reflecting on Ryan Coles lack of urgency on analysing the clots and lack of real world observations I'm starting to lean towards the naked emporers (Ivor Cumins) viewpoint that there's either fakery or it's not as frequent as some people would have us believe. Either way it needs investigating.
It takes considerable time to break down fibrin. If it is extensive like that - you wouldn’t live to dissolve it.
Perhaps. Martenson also does a back of envelope fibrin - to - blood volume calculation but that's in the assumption of post-mortem pooling, not of circulation + buildup.