Brian, I had to let you know that Omicron does in fact evade SARS-1 immunity. Ask me how I know and I'll see if I can explain with this headache and brain fog. With that, it was fairly mild. My wife is having a tougher time. I had SARS-1, wife stopped at 2 jabs.
Brian - Seriously, God bless you. You are doing such incredibly important work, laying out the biochemical architecture of what is happening on earth in similar fashion to Katherine Watt revealing the legal architecture. I do not fully understand all your analysis. But I understand enough to see how true your aim is and how committed you are to both accuracy and precision. Please know you are deeply appreciated.
Nov 17, 2022·edited Nov 17, 2022Liked by Brian Mowrey
So as mentioned in the SARS-COV1 study Sotrovimab was a monoclonal antibody that was isolated from someone previously infected with SARS-COV1. It was effective until Ba.2, likely from the T376A, D405N, and R408S group of mutations. These amino acids aren't on the surface, and so I believe it's assumed that these mutations led to a different folding pattern in the spike that shielded Sortrovimab's epitopes and stopped it from binding.
I don't understand this statement:
"Fusion allows the virus to spread from cell to cell without leaving the cell."
I thought that once a virus fused to a cell it effectively became part of the cell and injected its genetic payload into the cytosol .
Brian, I had to let you know that Omicron does in fact evade SARS-1 immunity. Ask me how I know and I'll see if I can explain with this headache and brain fog. With that, it was fairly mild. My wife is having a tougher time. I had SARS-1, wife stopped at 2 jabs.
Brian - Seriously, God bless you. You are doing such incredibly important work, laying out the biochemical architecture of what is happening on earth in similar fashion to Katherine Watt revealing the legal architecture. I do not fully understand all your analysis. But I understand enough to see how true your aim is and how committed you are to both accuracy and precision. Please know you are deeply appreciated.
So as mentioned in the SARS-COV1 study Sotrovimab was a monoclonal antibody that was isolated from someone previously infected with SARS-COV1. It was effective until Ba.2, likely from the T376A, D405N, and R408S group of mutations. These amino acids aren't on the surface, and so I believe it's assumed that these mutations led to a different folding pattern in the spike that shielded Sortrovimab's epitopes and stopped it from binding.
There is no “Omicron” because Covid is completely FAKE and virology is pure junk fake science.